Abstracts Submission | IAPNEOCON 2019

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Guidelines for Submission of Award & free papers

Research Paper Submission Guidelines

  1. The first author should be registered for the conference IAPNEOCON 2019.
  2. In the Gold Medal Award Category, the first author / presenting author should be a member of IAP Neonatology Chapter and of Central IAP.
  3. The research work should NOT be published before in any medical journal.
  4. Papers submitted for any AWARD category should not have been presented previously.
  5. Last date for Papers/Abstracts submission is 7 August 2019.

Prof. Meharban Singh Gold Medal Best Research Award

For award papers the “FULL” research paper should be submitted in word format not exceeding 2500 words described in following headings:

  1. i. Title page with 1. Title 2. Authors details 3. Corresponding authors address
  2. ii. Abstract (maximum 250 words) 1. Headings: Background, Methods, results and conclusion
  3. iii. Introduction
  4. iv. Study Methods
  5. v. Results
  6. vi. Strengths and limitations
  7. vii. Conclusions
  8. viii. References

The style of the manuscript should be as per Indian Pediatrics. The papers will be evaluated by 3 eminent Neonatologists. Top 5 papers will be selected for presentation at the conference in the AWARD category. The AWARD will be based on the combined score for paper and presentation.

Dr. Rekha Udani Best Free Paper Award

For Free paper, a structured abstract not more than 250 words should be submitted.
Abstract Structure: Introduction, Study Objective, Methods, Results and Conclusions.

Prof. Armida Fernandez Best Poster Award

For Free paper, a structured abstract not more than 250 words should be submitted.
Abstract Structure: Introduction, Study Objective, Methods, Results and Conclusions.

Template for Abstract Submission

Title: Title: Low vasopressin levels are associated with progression of sepsis to septic shock in preterm neonates of < 34 weeks’ gestation: a case matched cohort study.

Authors
Abhishek Somasekhara Aradhya, 1
Venkataseshan Sundaram, 1
Naresh Sachdeva, 2
Sourabh Dutta, 1
Shiv Sajan Saini, 1
Praveen kumar, 1

Affiliations: Division of Neonatology of Department of Pediatrics1 and Department of Endocrinology2, Post Graduate Institute of Medical Education and Research, Chandigarh, India.

ABSTRACT:

Background and objectives: Role of proteins and molecules associated with the progression of sepsis to septic shock is unclear in neonates. In this study, we have explored the association between arginine vasopressin (aVP) and inducible Nitric oxide synthase (iNOS) and progression of sepsis to septic shock in preterm neonates of < 34 weeks’ gestation and have generated an integrated clinico-metabolic-molecular model for prediction of such progression.

Methods:Consecutively born preterm neonates of < 34 weeks’ gestation with sepsis were included; those with major malformations or were moribund were excluded. Subjects were monitored for 7 days from onset of sepsis for features of septic shock. Inducible NOS and aVP levels were measured at the onset of sepsis as well as septic shock. Septic neonates without organ dysfunction acted as controls [matched for gestationand age at onset of sepsis]. Conditional stepwise logistic regression was done for model generation.

Results: Out of 104 neonates with sepsis 49 (47%) developed severe sepsis or septic shock. A clear progression time line from sepsis to septic shock was observed in 21 (20%) neonates while the remaining 28 neonates had features of organ dysfunction at presentation itself. The aVP levels (pg/ml) at the onset of sepsis were significantly low in those who progressed to septic shock in comparison to non-progressed septic neonates [median (IQR): 31 (2.5-80) vs. 99.8 (12-156); p=0.02]. Similarly, a non-significant increase in iNOS (IU/L) was observed in the progressed group [median (IQR): 3.6 (1.9-4.8) vs. 2.9 (1.4-4.6); p=0.3]. A model encompassing a pre-organ dysfunction need for mechanical ventilation [OR (95% C.I): 35.6 (3.8, 334); p=0.002] and low vasopressin levels [OR (95% C.I): 0.97 (0.96, 0.99); p=0.01] had the predictive ability of 89% for progression of sepsis to septic shock in this population.

Conclusions: Preterm septic neonates who progressed to septic shock had significantly suppressed vasopressin levels at baseline, contributing possibly to later vaso-dysregulation and shock. A larger study in similar population may identify the potential role of inducible nitric oxide synthase in sepsis progression.


Selection Process All abstracts will be evaluated anonymously and scored by the appropriate scientific committee members. Final decisions will be made by the Scientific Committee which will determine whether the abstract will be accepted or refused and if accepted, as:
- an oral presentation,
- a poster presentation